Skip NavigationSkip to Content

NCI at Frederick Scientific Publications Advanced Search

Search

  1. NCI-F Publications

  2. Return to Quick Search
Search
  2. Year Published:

Your search returned 7 results.
User Information
Export Records
  1. 1.   The CYP2E1-humanized transgenic mouse: Role of CYP2E1 in acetaminophen hepatotoxicity
  2. Cheung, C.; Yu, A. M.; Ward, J. M.; Krausz, K. W.; Akiyama, T. E.; Feigenbaum, L.; Gonzalez, F. J.
  3. Drug Metabolism and Disposition. 2005, MAR; 33(3): 449-457.
  1. 2.   Intra- and intercellular variations in the repair efficiency of O-6-methylguanine, and their contribution to kinetic complexity
  2. Souliotis, V. L.; Sfikakis, P. P.; Anderson, L. M.; Kyrtopoulos, S. A.
  3. Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis. 2004, DEC 21; 568(2): 155-170.
  1. 3.   Identification of oxidized mitochondria proteins in alcohol-exposed human hepatoma cells and mouse liver
  2. Suh, S. K.; Hood, B. L.; Kim, B. J.; Conrads, T. P.; Veenstra, T. D.; Song, B. J.
  3. Proteomics. 2004, NOV; 4(11): 3401-3412.
  1. 4.   Glycine N-methyltransferase tumor susceptibility gene in the benzo(a)pyrene-detoxification pathway
  2. Chen, S. Y.; Lin, J. R. V.; Darbha, R.; Lin, P. P.; Liu, T. Y.; Chen, Y. M. A.
  3. Cancer Research. 2004 64(10): 3617-3623.
  1. 5.   The drug salicylamide is an antagonist of the aryl hydrocarbon receptor that inhibits signal transduction induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin
  2. MacDonald, C. J.; Ciolino, H. P.; Yeh, G. C.
  3. Cancer Research. 2004 64(1): 429-434.
  1. 6.   Dibenzoylmethane modulates aryl hydrocarbon receptor function and expression of cytochromes P450 1A1, 1A2, and 1B1
  2. MacDonald, C. J.; Ciolino, H. P.; Yeh, G. C.
  3. Cancer Research. 2001 61(10): 3919-3924.
  1. 7.   Ethanol Down-Regulates the Transcription of Microsomal Triglyceride Transfer Protein Gene
  2. Lin, M. C. M.; Li, J. J.; Wang, E. J.; Princler, G. L.; Kauffman, F. C.; Kung, H. F.
  3. Faseb Journal. 1997 11(13): 1145-1152.
NCI at Frederick

You are leaving a government website.

This external link provides additional information that is consistent with the intended purpose of this site. The government cannot attest to the accuracy of a non-federal site.

Linking to a non-federal site does not constitute an endorsement by this institution or any of its employees of the sponsors or the information and products presented on the site. You will be subject to the destination site's privacy policy when you follow the link.

ContinueCancel