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  1. 1.   Sulindac regulates the aryl hydrocarbon receptor-mediated expression of Phase 1 metabolic enzymes in vivo and in vitro
  2. Ciolino, H. P.; MacDonald, C. J.; Memon, O. S.; Bass, S. E.; Yeh, G. C.
  3. Carcinogenesis. 2006, Aug; 27(8): 1586-1592.
  1. 2.   Provision of granulocyte-macrophage colony-stimulating factor converts an autoimmune response to a self-antigen into an antitumor response
  2. Ji, Q. Y.; Gondek, D.; Hurwitz, A. A.
  3. Journal of Immunology. 2005, AUG 1; 175(3): 1456-1463.
  1. 3.   Reactive oxygen species and lung tumorigenesis by mutant K-ras: A working hypothesis
  2. Maciag, A.; Anderson, L. M.
  3. Experimental Lung Research. 2005, JAN-FEB; 31(1): 83-104.
  1. 4.   Minimal influence of metallothionein over-expression on nickel carcinogenesis in mice
  2. Waalkes, M. P.; Liu, J.; Kasprzak, K. S.; Diwan, B. A.
  3. Toxicology Letters. 2004, NOV 28; 153(3): 357-364.
  1. 5.   Induction of tumors of the liver, lung, ovary and adrenal in adult mice after brief maternal gestational exposure to inorganic arsenic: promotional effects of postnatal phorbol ester exposure on hepatic and pulmonary, but not dermal cancers
  2. Waalkes, M. P.; Ward, J. M.; Diwan, B. A.
  3. Carcinogenesis. 2004 25(1): 133-141.
  1. 6.   Decrease in K-ras p21 and increase in Raf1 and activated Erk 1 and 2 in murine lung tumors initiated by N-nitrosodimethylamine and promoted by 2,3,7,8-tetrachlorodibenzo-p-dioxin
  2. Ramakrishna, G.; Perella, C.; Birely, L.; Diwan, B. A.; Fornwald, L. W.; Anderson, L. M.
  3. Toxicology and Applied Pharmacology. 2002 179(1): 21-34.
  1. 7.   K-ras mutations in mouse lung tumors of extreme age: independent of paternal preconceptional exposure to chromium(III) but significantly more frequent in carcinomas than adenomas
  2. McKenna, I. M.; Ramakrishna, G.; Diwan, B. A.; Shiao, Y. H.; Kasprzak, K. S.; Powell, D. A.; Anderson, L. M.
  3. Mutation Research-Genetic Toxicology and Environmental Mutagenesis. 2001 490(1): 57-65.
  1. 8.   Heterozygous inactivation of TGF-beta 1 increases the susceptibility to chemically induced mouse lung tumorigenesis independently of mutational activation of K-ras
  2. McKenna, I. M.; Ramakrishna, G.; Diwan, B. A.; Kang, Y.; Shiao, Y. H.; Wakefield, L. M.; Powell, D. A.; Anderson, L. M.; Jakowlew, S. B.
  3. Toxicology Letters. 2001 123(2-3): 151-158.
  1. 9.   Pathology of aging B6;129 mice
  2. Haines, D. C.; Chattopadhyay, S.; Ward, J. M.
  3. Toxicologic Pathology. 2001 29(6): 653-661.
  1. 10.   Selective Mutation of K-Ras By N-Ethylnitrosourea Shifts From Codon 12 to Codon 61 During Fetal Mouse Lung Maturation
  2. Sithanandam, G.; Ramakrishna, G.; Diwan, B. A.; Anderson, L. M.
  3. Oncogene. 1998 17(4): 493-502.
  1. 11.   Skin Tumorigenesis and Ki-Ras and Ha-Ras Mutations in Tumors From Adult Mice Exposed in Utero to 3'-Azido-2',3'-Dideoxythymidine
  2. Zhang, Z. H.; Diwan, B. A.; Anderson, L. M.; Logsdon, D.; Olivero, O. A.; Haines, D. C.; Rice, J. M.; Yuspa, S. H.; Poirier, M. C.
  3. Molecular Carcinogenesis. 1998 23(1): 45-51.
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