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  1. 1.   gamma-cleavage-independent functions of presenilin, nicastrin, and aph-1 regulate cell-junction organization and prevent tau toxicity in vivo
  2. Doglio, L. E.; Kanwar, R.; Jackson, G. R.; Perez, M.; Avila, J.; Dingwal, C.; Dotti, C. G.; Fortini, M. E.; Feiguin, F.
  3. Neuron. 2006, MAY 4; 50(3): 359-375.
  1. 2.   Progressive phenotype and nuclear accumulation of an amino-terminal cleavage fragment in a transgenic mouse model with inducible expression of full-length mutant huntingtin
  2. Tanaka, Y.; Igarashi, S.; Nakamura, M.; Gafni, J.; Torcassi, C.; Schilling, G.; Crippen, D.; Wood, J. D.; Sawa, A.; Jenkins, N. A.; Copeland, N. G.; Borchelt, D. R.; Ross, C. A.; Ellerby, L. M.
  3. Neurobiology of Disease. 2006, FEB; 21(2): 381-391.
  1. 3.   Parkinson's disease alpha-synuclein transgenic mice develop neuronal mitochondrial degeneration and cell death
  2. Martin, L. J.; Pan, Y.; Price, A. C.; Sterling, W.; Copeland, N. G.; Jenkins, N. A.; Price, D. L.; Lee, M. K.
  3. Journal of Neuroscience. 2006, JAN 4; 26(1): 41-50.
  1. 4.   3-Dimensional visualization of lesions in rat brain using magnetic resonance imaging microscopy
  2. Lester, D. S.; Lyon, R. C.; McGregor, G. N.; Engelhardt, R. T.; Schmued, L. C.; Johnson, G. A.; Johannessen, J. J.
  3. Neuroreport. 1999 10(4): 737-741.
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