Skip NavigationSkip to Content

NCI at Frederick Scientific Publications Advanced Search

Search

  1. NCI-F Publications

  2. Return to Quick Search
Search
  2. Year Published:

Your search returned 12 results.
User Information
Export Records
  1. 1.   Remodelin Is a Cryptic Assay Interference Chemotype That Does Not Inhibit NAT10-Dependent Cytidine Acetylation
  2. Shrimp,Jonathan; Jing,Yihang; Thalalla Gamage,Supuni; Nelson, Kathryn M.; Han, Joseph; Bryson, Keri M.; Montgomery,David; Thomas, Justin M.; Nance,Kellie; Sharma, Sunny; Fox,Stephen; Andresson,Thorkell; Sinclair,Wilson; Wu, Hong; Allali-Hassani, Abdellah; Senisterra, Guillermo; Vedadi, Masoud; Lafontaine, Denis; Dahlin, Jayme L.; Marmorstein, Ronen; Walters, Michael A.; Meier,Jordan
  3. ACS MEDICINAL CHEMISTRY LETTERS. 2021, Jun 10; 12(6): 887-892.
  1. 2.   Enhanced nuclear protein export in premature aging and rescue of the progeria phenotype by modulation of CRM1 activity
  2. García-Aguirre, Ian; Alamillo-Iniesta, Alma; Rodríguez-Pérez, Ruth; Vélez-Aguilera, Griselda; Amaro-Encarnación, Elianeth; Jiménez-Gutiérrez, Elizabeth; Vásquez-Limeta, Alejandra; Samuel Laredo-Cisneros, Marco; Morales-Lázaro, Sara L; Tiburcio-Félix, Reynaldo; Ortega, Arturo; Magaña, Jonathan J; Winder, Steve J; Cisneros, Bulmaro
  3. Aging cell. 2019, Jul 15; e13002.
  1. 3.   Functional Coupling between the Extracellular Matrix and Nuclear Lamina by Wnt Signaling in Progeria
  2. Hernandez, L.; Roux, K. J.; Wong, E. S. M.; Mounkes, L. C.; Mutalif, R.; Navasankari, R.; Rai, B.; Cool, S.; Jeong, J. W.; Wang, H. H.; Lee, H. S.; Kozlov, S.; Grunert, M.; Keeble, T.; Jones, C. M.; Meta, M. D.,; Young, S. G.; Daar, I. O.; Burke, B.; Perantoni, A. O.; Stewart, C. L.
  3. Developmental Cell. 2010, Sep 14; 19(3): 413-425.
  1. 4.   Nuclear lamin A/C deficiency induces defects in cell mechanics, polarization, and migration
  2. Lee, J. S. H.; Hale, C. M.; Panorchan, P.; Khatau, S. B.; George, J. P.; Tseng, Y.; Stewart, C. L.; Hodzic, D.; Wirtz, D.
  3. Biophysical Journal. 2007, Oct; 93(7): 2542-2552.
  1. 5.   Mouse models of the laminopathies
  2. Stewart, C. L.; Kozlov, S.; Fong, L. G.; Young, S. G.
  3. Experimental Cell Research. 2007, Jun; 313(10): 2144-2156.
  1. 6.   Lamins A and C but not lamin B1 regulate nuclear mechanics
  2. Lammerding, J.; Fong, L. G.; Ji, J. Y.; Reue, K.; Stewart, C. L.; Young, S. G.; Lee, R. T.
  3. Journal of Biological Chemistry. 2006, Sep; 281(35): 25768-25780.
  1. 7.   Prelamin A and lamin A appear to be dispensable in the nuclear lamina
  2. Fong, L. G.; Ng, J. K.; Lammerding, J.; Vickers, T. A.; Meta, M.; Cote, N.; Gavino, B.; Qiao, X.; Chang, S. Y.; Young, S. R.; Yang, S. H.; Stewart, C. L.; Lee, R. T.; Bennett, C. F.; Bergo, M. O.; Young, S. G.
  3. Journal of Clinical Investigation. 2006, MAR; 116(3): 743-752.
  1. 8.   The laminopathies: The functional architecture of the nucleus and its contribution to disease
  2. Burke, B.; Stewart, C. L.
  3. Annual review of genomics and human genetics. 2006 7: 369-405.
  1. 9.   Structural organization and functions of the nucleus in development, aging, and disease
  2. Mounkes, L.; Stewart, C. L.; Schatten, G. P.
  3. Current Topics in Developmental Biology. 2004; 61 : 191-228.
  1. 10.   Lamin A/C deficiency causes defective nuclear mechanics and mechanotransduction
  2. Lammerding, J.; Schulze, P. C.; Takahashi, T.; Kozlov, S.; Sullivan, T.; Kamm, R. D.; Stewart, C. L.; Lee, R. T.
  3. Journal of Clinical Investigation. 2004 113(3): 370-378.
  1. 11.   Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C-deficient mice
  2. Nikolova, V.; Leimena, C.; McMahon, A. C.; Tan, J. C.; Chandar, S.; Jogia, D.; Kesteven, S. H.; Michalicek, J.; Otway, R.; Verheyen, F.; Rainer, S.; Stewart, C. L.; Martin, D.; Feneley, M. P.; Fatkin, D.
  3. Journal of Clinical Investigation. 2004 113(3): 357-369.
  1. 12.   Aging and nuclear organization: lamins and progeria
  2. Mounkes, L. C.; Stewart, C. L.
  3. Current Opinion in Cell Biology. 2004 16(3): 322-327.
NCI at Frederick

You are leaving a government website.

This external link provides additional information that is consistent with the intended purpose of this site. The government cannot attest to the accuracy of a non-federal site.

Linking to a non-federal site does not constitute an endorsement by this institution or any of its employees of the sponsors or the information and products presented on the site. You will be subject to the destination site's privacy policy when you follow the link.

ContinueCancel