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  1. 1.   Loss of myeloid-specific lamin A/C drives lung metastasis through Gfi-1 and C/EBPe-mediated granulocytic differentiation
  2. Ishii, Hiroki; Park, Woo-Yong; So, Jaeyoung; Kuhn,Skyler; Koparde,Vishal; Pang, Yanli; Greten, Tim F; Hollander, M Christine; Yang, Li
  3. Molecular carcinogenesis. 2020, Jan 07;
  1. 2.   Behavioral and Molecular Exploration of the AR-CMT2A Mouse Model Lmna (R298C/R298C)
  2. Poitelon, Y.; Kozlov, S.; Devaux, J.; Vallat, J. M.; Jamon, M.; Roubertoux, P.; Rabarimeriarijaona, S.; Baudot, C.; Hamadouche, T.; Stewart, C. L.; Levy, N.; Delague, V.
  3. Neuromolecular Medicine. 2012, Mar; 14(1): 40-52.
  1. 3.   Mouse models of the laminopathies
  2. Stewart, C. L.; Kozlov, S.; Fong, L. G.; Young, S. G.
  3. Experimental Cell Research. 2007, Jun; 313(10): 2144-2156.
  1. 4.   Prelamin A and lamin A appear to be dispensable in the nuclear lamina
  2. Fong, L. G.; Ng, J. K.; Lammerding, J.; Vickers, T. A.; Meta, M.; Cote, N.; Gavino, B.; Qiao, X.; Chang, S. Y.; Young, S. R.; Yang, S. H.; Stewart, C. L.; Lee, R. T.; Bennett, C. F.; Bergo, M. O.; Young, S. G.
  3. Journal of Clinical Investigation. 2006, MAR; 116(3): 743-752.
  1. 5.   Nuclear lamin A inhibits adipocyte differentiation: implications for Dunnigan-type familial partial lipodystrophy
  2. Boguslavsky, R. L.; Stewart, C. L.; Worman, H. J.
  3. Human Molecular Genetics. 2006, FEB 15; 15(4): 653-663.
  1. 6.   Loss of emerin at the nuclear envelope disrupts the Rb1/E2F and MyoD pathways during muscle regeneration
  2. Melcon, G.; Kozlov, S.; Cutler, D. A.; Sullivan, T.; Hernandez, L.; Zhao, P.; Mitchell, S.; Nader, G.; Bakay, M.; Rottman, J. N.; Hoffman, E. P.; Stewart, C. L.
  3. Human Molecular Genetics. 2006, FEB 15; 15(4): 637-651.
  1. 7.   Dependence of diffusional mobility of integral inner nuclear membrane proteins on A-type lamins
  2. Ostlund, C.; Sullivan, T.; Stewart, C. L.; Worman, H. J.
  3. Biochemistry. 2006, FEB 7; 45(5): 1374-1382.
  1. 8.   The laminopathies: The functional architecture of the nucleus and its contribution to disease
  2. Burke, B.; Stewart, C. L.
  3. Annual review of genomics and human genetics. 2006 7: 369-405.
  1. 9.   Structural organization and functions of the nucleus in development, aging, and disease
  2. Mounkes, L.; Stewart, C. L.; Schatten, G. P.
  3. Current Topics in Developmental Biology. 2004; 61 : 191-228.
  1. 10.   Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C-deficient mice
  2. Nikolova, V.; Leimena, C.; McMahon, A. C.; Tan, J. C.; Chandar, S.; Jogia, D.; Kesteven, S. H.; Michalicek, J.; Otway, R.; Verheyen, F.; Rainer, S.; Stewart, C. L.; Martin, D.; Feneley, M. P.; Fatkin, D.
  3. Journal of Clinical Investigation. 2004 113(3): 357-369.
  1. 11.   The laminopathies: nuclear structure meets disease
  2. Mounkes, L.; Kozlov, S.; Burke, B.; Stewart, C. L.
  3. Current Opinion in Genetics & Development. 2003 13(3): 223-230.
  1. 12.   Characterization of adiposity and metabolism in Lmna-deficient mice
  2. Cutler, D. A.; Sullivan, T.; Marcus-Samuels, B.; Stewart, C. L.; Reitman, M. L.
  3. Biochemical and Biophysical Research Communications. 2002 291(3): 522-527.
  1. 13.   Nuclear envelope defects associated with LMNA mutations cause dilated cardiomyopathy and Emery-Dreifuss muscular dystrophy
  2. Raharjo, W. H.; Enarson, P.; Sullivan, T.; Stewart, C. L.; Burke, B.
  3. Journal of Cell Science. 2001 114(24): 4447-4457.
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