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  1. 1.   Inactivation of the mouse Magel2 gene results in growth abnormalities similar to Prader-Willi syndrome
  2. Bischof, J. M.; Stewart, C. L.; Wevrick, R.
  3. Human Molecular Genetics. 2007, Nov; 16(22): 2713-2719.
  1. 2.   The imprinted gene Magel2 regulates normal circadian output
  2. Kozlov, S. V.; Bogenpohl, J. W.; Howell, M. P.; Wevrick, R.; Panda, S.; Hogenesch, J. B.; Muglia, L. J.; Van Gelder, R. N.; Herzog, E. D.; Stewart, C. L.
  3. Nature Genetics. 2007, Oct; 39(10): 1266-1272.
  1. 3.   Absence of Ndn, encoding the Prader-Willi syndrome-deleted gene necdin, results in congenital deficiency of central respiratory drive in neonatal mice
  2. Ren, J.; Lee, S.; Pagliardini, S.; Gerard, M.; Stewart, C. L.; Greer, J. J.; Wevrick, R.
  3. Journal of Neuroscience. 2003 23(5): 1569-1573.
  1. 4.   Expression and imprinting of MAGEL2 suggest a role in Prader-Willi syndrome and the homologous murine imprinting phenotype
  2. Lee, S.; Kozlov, S.; Hernandez, L.; Chamberlain, S. J.; Brannan, C. I.; Stewart, C. L.; Wevrick, R.
  3. Human Molecular Genetics. 2000 9(12): 1813-1819.
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