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  1. 1.   CHEK2 genomic and proteomic analyses reveal genetic inactivation or endogenous activation across the 60 cell lines of the US National Cancer Institute
  2. Zoppoli, G.; Solier, S.; Reinhold, W. C.; Liu, H.; Connelly, J. W.; Monks, A.; Shoemaker, R. H.; Abaan, O. D.; Davis, S. R.; Meltzer, P. S.; Doroshow, J. H.; Pommier, Y.
  3. Oncogene. 2012, Jan; 31(4): 403-418.
  1. 2.   Phosphatidylinositol ether lipid analogues that inhibit AKT also independently activate the stress kinase, p38 alpha, through MKK3/6-independent and -dependent mechanisms
  2. Gills, J. J.; Castillo, S. S.; Zhang, C. Y.; Petukhov, P. A.; Memmott, R. M.; Hollingshead, M.; Warfel, N.; Han, J. H.; Kozikowski, A. P.; Dennis, P. A.
  3. Journal of Biological Chemistry. 2007, Sep; 282(37): 27020-27029.
  1. 3.   Inactivation of p53 by human T-cell lymphotropic virus type 1 tax requires activation of the NF-kappa B pathway and is dependent on p53 phosphorylation
  2. Pise-Masison, C. A.; Mahieux, R.; Jiang, H.; Ashcroft, M.; Radonovich, M.; Duvall, J.; Guillerm, C.; Brady, J. N.
  3. Molecular and Cellular Biology. 2000 20(10): 3377-3386.
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