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  1. 1.   Non-B DNA-forming Sequences and WRN Deficiency Independently Increase the Frequency of Base Substitution in Human Cells
  2. Bacolla, A.; Wang, G. L.; Jain, A.; Chuzhanova, N. A.; Cer, R. Z.; Collins, J. R.; Cooper, D. N.; Bohr, V. A.; Vasquez, K. M.
  3. Journal of Biological Chemistry. 2011, Mar; 286(12): 10017-10026.
  1. 2.   DNA damage, superoxide, and mutant K-ras in human lung adenocarcinoma cells
  2. Romanowska, M.; Maciag, A.; Smith, A. L.; Fields, J. R.; Fornwald, L. W.; Kikawa, K. D.; Kasprzak, K. S.; Anderson, L. M.
  3. Free Radical Biology and Medicine. 2007, Oct; 43(8): 1145-1155.
  1. 3.   Inhibition of prolidase activity by nickel causes decreased growth of proline auxotrophic CHO cells
  2. Miltylk, W.; Surazynski, A.; Kasprzak, K. S.; Fivash, M. J.; Buzard, G. S.; Phang, J. M.
  3. Journal of Cellular Biochemistry. 2005, APR 15; 94(6): 1210-1217.
  1. 5.   The octapeptidic end of the C-terminal tail of histone H2A is cleaved off in cells exposed to carcinogenic nickel(II)
  2. Karaczyn, A. A.; Bal, W.; North, S. L.; Bare, R. M.; Hoang, V. M.; Fisher, R. J.; Kasprzak, K. S.
  3. Chemical Research in Toxicology. 2003 16(12): 1555-1559.
  1. 6.   Induction of oxidative DNA damage by carcinogenic metals
  2. Bal, W.; Kasprzak, K. S.
  3. Toxicology Letters. 2002 127(1-3): 55-62.
  1. 7.   Oxidative DNA and protein damage in metal-induced toxicity and carcinogenesis
  2. Kasprzak, K. S.
  3. Free Radical Biology and Medicine. 2002 32(10): 958-967.
  1. 8.   Mechanisms of arsenic-induced cross-tolerance to nickel cytotoxicity, genotoxicity, and apoptosis in rat liver epithelial cells
  2. Qu, W.; Kasprzak, K. S.; Kadiiska, M.; Liu, J.; Chen, H.; Maciag, A.; Mason, R. P.; Waalkes, M. P.
  3. Toxicological Sciences. 2001 63(2): 189-195.
  1. 9.   L-tyrosine and nitric oxide synergize to prevent cytotoxic effects of superoxide
  2. Souici, A. C.; Fitzhugh, A. L.; Keefer, L. K.; Felley-Bosco, E.
  3. Toxicology. 2001 165(2-3): 163-170.
  1. 11.   Transition mutation in codon 248 of the p53 tumor suppressor gene induced by reactive oxygen species and a nitric oxide-releasing compound
  2. Souici, A. C.; Mirkovitch, J.; Hausel, P.; Keefer, L. K.; Felley-Bosco, E.
  3. Carcinogenesis. 2000 21(2): 281-287.
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