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  1. 1.   A role for presenilin in post-stress regulation: effects of presenilin mutations on Ca2+ currents in Drosophila
  2. Lu, Y. S.; Lv, Y. B.; Ye, Y. H.; Wang, Y. L.; Hong, Y.; Fortini, M. E.; Zhong, Y.; Xie, Z. P.
  3. Faseb Journal. 2007, Aug; 21(10): 2368-2378.
  1. 2.   gamma-cleavage-independent functions of presenilin, nicastrin, and aph-1 regulate cell-junction organization and prevent tau toxicity in vivo
  2. Doglio, L. E.; Kanwar, R.; Jackson, G. R.; Perez, M.; Avila, J.; Dingwal, C.; Dotti, C. G.; Fortini, M. E.; Feiguin, F.
  3. Neuron. 2006, MAY 4; 50(3): 359-375.
  1. 3.   Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretase
  2. Jankowsky, J. L.; Fadale, D. J.; Anderson, J.; Xu, G. M.; Gonzales, V.; Jenkins, N. A.; Copeland, N. G.; Lee, M. K.; Younkin, L. H.; Wagner, S. L.; Younkin, S. G.; Borchelt, D. R.
  3. Human Molecular Genetics. 2004 13(2): 159-170.
  1. 4.   Effect of caspase cleavage-site phosphorylation on proteolysis
  2. Tozser, J.; Bagossi, N.; Zahuczky, G.; Specht, S. I.; Majerova, E.; Copeland, T. D.
  3. Biochemical Journal. 2003 372(Part 1): 137-143.
  1. 5.   Different cofactor activities in gamma-secretase assembly: evidence for a nicastrin-Aph-1 subcomplex
  2. Hu, Y.; Fortini, M. E.
  3. Journal of Cell Biology. 2003 161(4): 685-690.
  1. 6.   Double trouble for neurons
  2. Fortini, M. E.
  3. Nature. 2003 425(6958): 565-566.
  1. 7.   Human neurodegenerative disease modeling using Drosophila
  2. Bonini, N. M.; Fortini, M. E.
  3. Annual Review of Neuroscience. 2003 26: 627-656.
  1. 8.   Accumulation of proteolytic fragments of mutant presenilin 1 and accelerated amyloid deposition are co-regulated in transgenic mice
  2. Borchelt, D. R.; Lee, M. K.; Gonzales, V.; Slunt, H. H.; Ratovitski, T.; Jenkins, N. A.; Copeland, N. G.; Price, D. L.; Sisodia, S. S.
  3. Neurobiology of Aging. 2002 23(2): 171-177.
  1. 9.   Human alpha-synuclein-harboring familial Parkinson's disease- linked Ala-53 -> Thr mutation causes neurodegenerative disease with alpha-synuclein aggregation in transgenic mice
  2. Lee, M. K.; Stirling, W.; Xu, Y. Q.; Xu, X. Y.; Qui, D.; Mandir, A. S.; Dawson, T. M.; Copeland, N. G.; Jenkins, N. A.; Price, D. L.
  3. Proceedings of the National Academy of Sciences of the United States of America. 2002 99(13): 8968-8973.
  1. 10.   The central role of the trans-golgi network as a gateway of the early secretory pathway: Physiologic vs nonphysiologic protein transit
  2. Tekirian, T. L.
  3. Experimental Cell Research. 2002 281(1): 9-18.
  1. 11.   gamma-secretase-mediated proteolysis in cell-surface-receptor signalling
  2. Fortini, M. E.
  3. Nature Reviews Molecular Cell Biology. 2002 3(9): 673-684.
  1. 12.   Co-expression of multiple transgenes in mouse CNS: a comparison of strategies
  2. Jankowsky, J. L.; Slunt, H. H.; Ratovitski, T.; Jenkins, N. A.; Copeland, N. G.; Borchelt, D. R.
  3. Biomolecular Engineering. 2001 17(6): 157-165.
  1. 13.   Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin
  2. Schilling, G.; Becher, M. W.; Sharp, A. H.; Jinnah, H. A.; Duan, K.; Kotzuk, J. A.; Slunt, H. H.; Ratovitski, T.; Cooper, J. K.; Jenkins, N. A.; Copeland, N. G.; Price, D. L.; Ross, C. A.; Borchelt, D. R.
  3. Human Molecular Genetics. 1999 8(3): 397-407.
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