Skip NavigationSkip to Content

NCI at Frederick Scientific Publications Advanced Search

Search

  1. NCI-F Publications

  2. Return to Quick Search
Search
  2. Year Published:

Your search returned 12 results.
User Information
Export Records
  1. 1.   Bacterial lipopolysaccharide selectively up-regulates the function of the chemotactic peptide receptor formyl peptide receptor 2 in murine microglial cells
  2. Cui, Y. H.; Le, Y. Y.; Gong, W. H.; Proost, P.; Van Damme, J.; Murphy, W. J.; Wang, J. M.
  3. Journal of Immunology. 2002 168(1): 434-442.
  1. 2.   Human dendritic cells express functional formyl peptide receptor-like-2 (FPRL2) throughout maturation
  2. Yang, D.; Chen, Q.; Gertz, B.; He, R.; Phulsuksombati, M.; Ye, R. D.; Oppenheim, J. J.
  3. Journal of Leukocyte Biology. 2002 72(3): 598-607.
  1. 3.   Differential regulation of formyl peptide receptor-like 1 expression during the differentiation of monocytes to dendritic cells and macrophages
  2. Yang, D.; Chen, Q.; Le, Y. Y.; Wang, J. M.; Oppenheim, J. J.
  3. Journal of Immunology. 2001 166(6): 4092-4098.
  1. 4.   Synthetic peptide MMK-1 is a highly specific chemotactic agonist for leukocyte FPRL1
  2. Hu, J. Y.; Le, Y. Y.; Gong, W. H.; Dunlop, N. M.; Gao, J. L.; Murphy, P. M.; Wang, J. M.
  3. Journal of Leukocyte Biology. 2001 70(1): 155-161.
  1. 5.   Characterization of chenodeoxycholic acid as an endogenous antagonist of the G-coupled formyl peptide receptors
  2. Chen, X.; Yang, D.; Shen, W.; Dong, H. F.; Wang, J. M.; Oppenheim, J. J.; Howard, O. M. Z.
  3. Inflammation Research. 2000 49(12): 744-755.
  1. 6.   Activation of the chemotactic peptide receptor FPRL1 in monocytes phosphorylates the chemokine receptor CCR5 and attenuates cell responses to selected chemokines
  2. Shen, W. P.; Proost, P.; Li, B. Q.; Gong, W. H.; Le, Y. Y.; Sargeant, R.; Murphy, P. M.; Van Damme, J.; Wang, J. M.
  3. Biochemical and Biophysical Research Communications. 2000 272(1): 276-283.
  1. 7.   N36, a synthetic N-terminal heptad repeat domain of the HIV-1 envelope protein gp41, is an activator of human phagocytes
  2. Le, Y. Y.; Jiang, S.; Hu, J. Y.; Gong, W. H.; Su, S. B.; Dunlop, N. M.; Shen, W. P.; Li, B. Q.; Wang, J. M.
  3. Clinical Immunology. 2000 96(3): 236-242.
  1. 8.   Novel pathophysiological role of classical chemotactic peptide receptors and their communications with chemokine receptors
  2. Le, Y. Y.; Li, B. Q.; Gong, W. H.; Shen, W. P.; Hu, J. Y.; Dunlop, N. M.; Oppenheim, J. J.; Wang, J. M.
  3. Immunological Reviews. 2000 177: 185-194.
  1. 9.   New insights into the biology of the acute phase response
  2. Suffredini, A. F.; Fantuzzi, G.; Badolato, R.; Oppenheim, J. J.; O'Grady, N. P.
  3. Journal of Clinical Immunology. 1999 19(4): 203-214.
  1. 10.   Utilization of two seven-transmembrane, G protein-coupled receptors, formyl peptide receptor-like 1 and formyl peptide receptor, by the synthetic hexapeptide WKYMVm for human phagocyte activation
  2. Le, Y. Y.; Gong, W. H.; Li, B. Q.; Dunlop, N. M.; Shen, W. P.; Su, S. B.; Ye, R. D.; Wang, J. M.
  3. Journal of Immunology. 1999 163(12): 6777-6784.
  1. 11.   T21/DP107, a synthetic leucine zipper-like domain of the HIV-1 envelope gp41, attracts and activates human phagocytes by using G-protein-coupled formyl peptide receptors
  2. Su, S. B.; Gao, J. L.; Gong, W. H.; Dunlop, N. M.; Murphy, P. M.; Oppenheim, J. J.; Wang, J. M.
  3. Journal of Immunology. 1999 162(10): 5924-5930.
  1. 12.   A synthetic peptide derived from human immunodeficiency virus type 1 gp120 downregulates the expression and function of chemokine receptors CCR5 and CXCR4 in monocytes by activating the 7-transmembrane G-protein-coupled receptor FPRL1/LXA4R
  2. Deng, X. Y.; Ueda, H.; Su, S. B.; Gong, W. H.; Dunlop, N. M.; Gao, J. L.; Murphy, P. M.; Wang, J. M.
  3. Blood. 1999 94(4): 1165-1173.
NCI at Frederick

You are leaving a government website.

This external link provides additional information that is consistent with the intended purpose of this site. The government cannot attest to the accuracy of a non-federal site.

Linking to a non-federal site does not constitute an endorsement by this institution or any of its employees of the sponsors or the information and products presented on the site. You will be subject to the destination site's privacy policy when you follow the link.

ContinueCancel